|
PMID
|
Match String
|
Actual String
|
Score
|
Flanking text
|
Edited by
|
Edit
|
| 15031734 | c-Jun | c-Jun | 1.3 | HNE activates c-Jun aminoterminal kinases and mitogen-activated protein kinase-1 (also also known as | |  |
| 15896810 | AP-1 | AP-1 | 1.8 | and/or and or DNA binding of numerous transcription factors including AP-1 fos jun myc erg-1 SAPK and NFkB 3 | | |
| 15896810 | JUN | JUN | 1.8 | to inhibit caspase-3 activation caused by ceramide and also affect JUN kinase and p38-kinase activation 141 | | |
| 16406002 | c-Jun | c-Jun | 1.3 | In various cell lines reactive aldehyde-induced apoptosis was accompanied by c-Jun -N-terminal kinase and caspase-3 activation | | |
| 17150307 | AP-1 | AP-1 | 2.8 | In addition GSH depletion enhanced oxidative stress markers AP-1 transcriptional activation c-Jun c-Fos and HO-1 expression in NSC34 cells | | |
| 17150307 | c-Jun | c-Jun | 2.8 | addition GSH depletion enhanced oxidative stress markers AP-1 transcriptional activation c-Jun c-Fos and HO-1 expression in NSC34 cells analyzed by a | | |
| 17150307 | AP-1 | AP-1 | 2.8 | EA treatment enhanced AP-1 transcriptional activation as detected by a luciferase reporter gene assay | | |
| 17150307 | c-Jun | c-Jun | 2.8 | detected by a luciferase reporter gene assay ( Figure 3A c-Jun ( Figure 3B and c-Fos expression (data data not shown | | |
| 17150307 | c-Jun | c-Jun | 2.8 | depletion increased the expression of the early oxidative stress markers c-Jun c-Fos (data data not shown and AP-1 activation ( Figure | | |
| 17150307 | AP-1 | AP-1 | 2.8 | oxidative stress markers c-Jun c-Fos (data data not shown and AP-1 activation ( Figure 3 and secondary oxidative stress markers such | | |
| 17191135 | AP-1 | AP-1 | 1.6 | HO-1 gene such as Fos/Jun Fos Jun activator protein-1 (AP-1)], AP-1 nuclear factor-kB (NFkB) NFkB and the more recently identified Nrf2 | | |
| 17191135 | AP-1 | AP-1 | 1.6 | and WT-ZFP conferring constitutive expression inducible expression elements such as AP-1 AP-2 NF-kB Oct-1 PEA-3 Myb and the antioxidant-responsive element (ARE) | | |
| 17191135 | AP-1 | AP-1 | 1.6 | DNA binding activity of critical transcription factors such as the AP-1 family members NF-kB Jun Fos p53 CREB PEBP2/CBF, PEBP2 CBF | | |
| 17634371 | c-Jun | c-Jun | 1.3 | is not completely elucidated but is associated with activation of c-Jun N-terminal kinase (JNK), JNK release of cytochrome c from mitochondria | | |
| 15031734 | c jun | c jun | 1.0 | hne activates c jun aminoterminal kinases and mitogen activated protein kinase 1 also known as p38 thereby stimulating an apoptotic cascade 15 . | | |
| 16406002 | c jun | c jun | 1.0 | in various cell lines reactive aldehyde induced apoptosis was accompanied by c jun n terminal kinase and caspase 3 activation. | | |
| 17150307 | c jun | c jun | 1.0 | in addition gsh depletion enhanced oxidative stress markers ap 1 transcriptional activation c jun c fos and ho 1 expression in nsc34 cells analyzed by a luciferase reporter western blotting and quantitative pcr assays respectively. | | |
| 17150307 | c jun | c jun | 1.0 | ea treatment enhanced ap 1 transcriptional activation as detected by a luciferase reporter gene assay figure 3a c jun figure 3b and c fos expression data not shown as detected by western analysis. | | |
| 17150307 | c jun | c jun | 1.0 | in addition gsh depletion increased the expression of the early oxidative stress markers c jun c fos data not shown and ap 1 activation figure 3 and secondary oxidative stress markers such as ho 1 expression figure 4 . | | |
| 17634371 | c jun | c jun | 1.0 | the death pathway is not completely elucidated but is associated with activation of c jun n terminal kinase jnk release of cytochrome c from mitochondria and subsequent activation of caspases 9 6 and 3 casaccia bonnefil et al. 1996 ; yoon et al. 1998 ; wang et al. 2001 ; bhakar et al. 200 | | |