|
PMID
|
Match String
|
Actual String
|
Score
|
Flanking text
|
Edited by
|
Edit
|
| 17191135 | ACTH | ACTH | 1.0 | endocrine response to a stressor stimulating the release of pituitary ACTH 44 | |  |
| 17597167 | ACTH | ACTH | 1.5 | the release of corticotrophin-releasing factor (CRF), CRF adrenocorticotrophic hormone (ACTH) ACTH and corticosterone | | |
| 17597167 | MSH | MSH | 1.5 | Fig 2 co-localised with _amp_#x3b1 -melanocyte stimulating hormone (_amp_#x3b1;-MSH) _amp_#x3b1 -MSH 45 whereas both IL-1ra and IL-1RI were found to be | | |
| 17597167 | MSH | MSH | 1.5 | The neuropeptide _amp_#x3b1 -MSH is known to have anti-pyretic and antiinflammatory properties (see see | | |
| 17597167 | MSH | MSH | 1.5 | Co-administration of _amp_#x3b1 -MSH and KA in rats showed a potentiating effect of _amp_#x3b1 | | |
| 17597167 | MSH | MSH | 1.5 | and KA in rats showed a potentiating effect of _amp_#x3b1 -MSH on KA-induced hyperthermia as well as on the initial KA-induced | | |
| 17597167 | MSH | MSH | 1.5 | well as on the initial KA-induced hypothermic effect whereas _amp_#x3b1 -MSH administered alone produced a dose-dependent increase in core temperature with | | |
| 17597167 | MSH | MSH | 1.5 | 42 47 51 and 53 see 41 and that _amp_#x3b1 -MSH has been shown to reduce the levels of IL-1 and | | |
| 17597167 | MSH | MSH | 1.5 | modulation of core temperature changes by KA 50 and _amp_#x3b1 -MSH respectively indicates the complexity of temperature regulation | | |
| 17597167 | MSH | MSH | 1.5 | A more consistent effect of _amp_#x3b1 -MSH on body temperature was observed in global cerebral ischaemia in | | |
| 17597167 | MSH | MSH | 1.5 | Thus pretreatment with _amp_#x3b1 -MSH resulted in a hypothermic response with continued decrease during the | | |
| 17597167 | MSH | MSH | 1.5 | In addition systemic administration of _amp_#x3b1 -MSH was found to potentiate the decrease in brain temperature observed | | |
| 17597167 | MSH | MSH | 1.5 | Several studies have shown that _amp_#x3b1 -MSH can provide protection against tissue damage due to ischaemia in | | |
| 17597167 | MSH | MSH | 1.5 | and reperfusion in the rat upon peripheral administration of _amp_#x3b1 -MSH 62 and in the Mongolian gerbil upon administration of a | | |
| 17597167 | MSH | MSH | 1.5 | Thus we found that upon post-ischaemic administration of _amp_#x3b1 -MSH there was a 75% larger number of viable neurons in | | |
| 17597167 | MSH | MSH | 1.5 | Neuroprotective and neurotrophic activities of _amp_#x3b1 -MSH have also been shown in various models of neuronal injury | | |
| 17597167 | MSH | MSH | 1.5 | Recently we have found that _amp_#x3b1 -MSH may provide neuroprotection also upon KA-induced excitotoxicity 65 | | |
| 17597167 | MSH | MSH | 1.5 | Also in this case was _amp_#x3b1 -MSH administered after the toxic insult and the rescuing effect seen | | |
| 17597167 | MSH | MSH | 1.5 | It is conceivable that the antiinflammatory properties of _amp_#x3b1 -MSH are responsible for its neuroprotective activities but other properties such | | |
| 17597167 | MSH | MSH | 1.5 | during global cerebral ischaemia in rats following pretreatment with _amp_#x3b1 -MSH ( n = 7 but not saline ( n = | | |
| 17597167 | MSH | MSH | 1.5 | during cerebral ischaemia is larger in rats pretreated with _amp_#x3b1 -MSH than after saline pretreatment ( p _amp_#x3c 0.05 | | |
| 17597167 | MSH | MSH | 1.5 | cerebral ischaemia/reperfusion ischaemia reperfusion followed by systemic administration of _amp_#x3b1 -MSH | | |
| 17597167 | MSH | MSH | 1.5 | Note the marked protection provided by post-ischaemic administration of _amp_#x3b1 -MSH (C) C | | |