HUGO ID Detailed Result 11892

PMID	Match String	Actual String	Score	Flanking text	Edited by	Edit
12654515	TNF	TNF-A	1.2	displayed a dose-dependent resistance to oxidative damage from hydrogen peroxide TNF-_amp_#x3b1 and mutant G93A-SOD1
12654515	TNF	TNF-A	1.2	TNF-_amp_#x3b1 N -acetyl-cysteine (N-AC), N-AC 5_amp_#x2032 5_amp_#x2032 -dimethylpryrroline-N -oxide (DMPO), DMPO
12654515	TNF	TNF-A	1.2	neuron-like cell death similar to that of hydrogen peroxide and TNF-_amp_#x3b1 ( Fig 1D 3.2
12654515	TNF	TNF-A	1.2	were infected with adenovirus containing mutant G93A-SOD1 and exposed to TNF-_amp_#x3b1 and hydrogen peroxide respectively
12654515	TNF	TNF-A	1.2	significant increases in cell survival in the presence of mSOD1 TNF-_amp_#x3b1 and hydrogen peroxide ( Fig 2 3.3
12654515	TNF	TNF-A	1.2	with WT-SOD1 mutant G93A-SOD1 or treated with hydrogen peroxide or TNF-_amp_#x3b1
12654515	TNF	TNF-A	1.2	A except that cells were treated with different concentrations of TNF-_amp_#x3b1
14739060	TNF	TNF	0.3	amplification resulting in production of neurotoxins such as cytokine (TNF, TNF IL1 eicosanoides ROS and RNS 9 and 13
15896810	TNF	TNF	1.2	It also protects against Fas-Apo-1 staurosporine TNF and etoposside-induced apoptotic cell death as well as H 2
16043017	TNF	TNF-	1.2	induced by SOD1 mutation 32 alteration of tumor necrosis factor TNF- and TNF--modulating cytokines 38 and 46
16043017	TNF	TNF--modulating	1.2	SOD1 mutation 32 alteration of tumor necrosis factor TNF- and TNF--modulating cytokines 38 and 46
16194581	TNF	TNF-A	1.2	synthase (iNOS), iNOS interleukin-1_amp_#x3b2 (IL-1_amp_#x3b2;), IL-1_amp_#x3b2 tumor necrosis factor-_amp_#x3b1 (TNF-_amp_#x3b1;), TNF-_amp_#x3b1 and nuclear factor-kappa B (NF-_amp_#x3ba;B) NF-_amp_#x3ba B 1 49 60
17496232	TNF-alpha	TNF-alpha	0.8	II gene expression is modulated by inflammatory mediators like cytokines TNF-alpha IFN-gamma and LPS that activate transcription factors like NF-kappaB or
17496232	TNF-alpha	TNF-alpha	0.8	In the extrinsic pathway binding of membrane ligands like TNF-alpha and Fas to membrane receptors triggers the activation of caspases
18414597	TNF-alpha	TNF-alpha	1.5	Pro-inflammatory cytokines (TNF-alpha TNF-alpha and IL-1 secretory phospholipase A(2) A 2 IIA and lipoprotein-PLA(2)
18414597	TNF-alpha	TNF-alpha	1.5	TNF-alpha and IL-1 alter lipid metabolism and stimulate production of eicosanoids
18422522	TNF-alpha	TNF-alpha	1.8	by these cells was activated by tumour necrosis factor-alpha (TNF-alpha), TNF-alpha a proinflammatory cytokine that plays important roles in ALS2 and
18422522	TNF-alpha	TNF-alpha-dependent	1.8	The TNF-alpha-dependent eNOS activation occurred through generation by sphingosine-kinase-1 of sphingosine-1-phosphate stimulation
18422522	TNF-alpha	TNF-alpha	1.8	constructs specific for the enzymes and receptors eNOS activation by TNF-alpha conferred cytoprotection from excitotoxicity and neurotoxic cues such as reactive
18751914	TNF	TNF-_amp_#945	0.1	Pro-inflammatory cytokines (TNF-_amp_#945; TNF-_amp_#945 and IL-1 secretory phospholipase A 2 IIA and lipoprotein-PLA 2
18751914	TNF	TNF-_amp_#945	0.1	TNF-_amp_#945 and IL-1 alter lipid metabolism and stimulate production of eicosanoids
12663085	tumor necrosis factor	tumor necrosis factor	1.0	a_amp_#x3b2; induces the production of tumor necrosis factor _amp_#x3b1; by microglia.
15896810	tumor necrosis factor	tumor necrosis factor	1.0	activation of inos requires gene transcription and the induction can be influenced by endotoxin and cytokines interleukin 1 interleukin 2 lipopolysaccharide interferon _amp_#x3b3; tumor necrosis factor .
15896810	tumor necrosis factor	tumor necrosis factor	1.0	it is involved in targeting and chaperoning of proteins degraded in proteasomes which include nfkb cyclins hsfs hypoxia inducible factor some apoptosis related proteins tumor necrosis factor and erythropoietin receptors [146] .
16043017	tumor necrosis factor	tumor necrosis factor	1.0	moreover free radical production in the g93a sod1 transgenic animals is induced by sod1 mutation [32] alteration of tumor necrosis factor tnf and tnf modulating cytokines [38] and [46] .
16194581	tumor necrosis factor	tumor necrosis factor	1.0	neuroinflammation has been connected to enhanced signal transduction leading to the activation genes such as the inducible nitric oxide synthase inos interleukin 1_amp_#x3b2; il 1_amp_#x3b2; tumor necrosis factor _amp_#x3b1; tnf _amp_#x3b1; and nuclear factor kappa b nf _amp_#x3ba;b [1] [49] [60] [65] [69] [72] [81] and [86] .
17496232	tnf alpha	tnf alpha	1.0	in contrast nos ii is not constitutive and does not depend on ca concentration; nos ii gene expression is modulated by inflammatory mediators like cytokines tnf alpha ifn gamma and lps that activate transcription factors like nf kappab or ap 1 59 .
17496232	tnf alpha	tnf alpha	1.0	in the extrinsic pathway binding of membrane ligands like tnf alpha and fas to membrane receptors triggers the activation of caspases and the induction of apoptosis 6 106 .
18414597	tnf alpha	tnf alpha	1.0	pro inflammatory cytokines tnf alpha and il 1 secretory phospholipase a 2 iia and lipoprotein pla 2 are implicated in vascular inflammation.
18414597	tnf alpha	tnf alpha	1.0	tnf alpha and il 1 alter lipid metabolism and stimulate production of eicosanoids ceramide and reactive oxygen species that potentiate cns injuries and certain neurological disorders.
18422522	tnf alpha	tnf alpha	1.0	we found that enos which is endogenously expressed by these cells was activated by tumour necrosis factor alpha tnf alpha a proinflammatory cytokine that plays important roles in als2 and several neurodegenerative diseases.
18422522	tnf alpha	tnf alpha	1.0	the tnf alpha dependent enos activation occurred through generation by sphingosine kinase 1 of sphingosine 1 phosphate stimulation of its membrane receptors and activation of akt as determined using small interfer
18422522	tnf alpha	tnf alpha	1.0	hate stimulation of its membrane receptors and activation of akt as determined using small interference rna and dominant negative constructs specific for the enzymes and receptors. enos activation by tnf alpha conferred cytoprotection from excitotoxicity and neurotoxic cues such as reactive oxygen species endoplasmic reticulum stress dna damage and mutated alsin itself.