HUGO ID Detailed Result 9595

PMID	Match String	Actual String	Score	Flanking text	Edited by	Edit
16647138	EP3	EP3	0.6	EP1 and EP3 agonists had little effect on membrane currents confirming
16647138	EP3	EP3	0.6	EP1 and EP3 agonists had little effect on membrane currents confirming that an
16647138	EP3	EP3	0.6	(1998) 1998 suggested an involvement of the EP3 receptor in febrile responses
16647138	EP3	EP3-deficient	0.6	They demonstrated that EP3-deficient mice failed to show a febrile response to intracerebroventricular injections
16647138	EP3	EP3	0.6	to those seen in control mice establishing that the cerebral EP3 receptors are involved in PGE 2 -evoked febrile responses
16647138	EP3	EP3	0.6	note that in addition to IP mRNA mRNAs for EP1 EP3 and EP4 were expressed in about 30% 50% and 20%
16647138	EP3	EP3	0.6	50% and 20% of the IP-receptor-positive neurons coexpressing the EP1 EP3 and EP4 receptor respectively ( Oida et al. 1995
17574754	EP3	EP3	0.3	receptor family where there are four subtypes (EP1, EP1 EP2 EP3 and EP4 and for the PGD 2 DP family where
17574754	EP3	EP3	0.3	with NMDA or oxygen glucose deprivation 16 and both the EP2 and EP3 receptors rescue motor neurons in organotypic spinal cord
17574754	EP3	EP3	0.3	or oxygen glucose deprivation 16 and both the EP2 and EP3 receptors rescue motor neurons in organotypic spinal cord slices subjected
17574754	EP3	EP3	0.3	and DP1 neuroprotection are dependent on intact cAMP signaling whereas EP3 neuroprotection is associated with increased AKT phosphorylation 3 and 16
17574754	EP3	EP3	0.3	previous studies we determined that PGE 2 signaling via its EP2 and EP3 receptors paradoxically protected motor neurons in a spinal
17574754	EP3	EP3	0.3	we determined that PGE 2 signaling via its EP2 and EP3 receptors paradoxically protected motor neurons in a spinal cord model
17574754	EP3	EP3	0.3	The EP3 agonist sulprostone exerted significant protection at pM_amp_#x2013 nM concentrations (
17574754	EP3	EP3	0.3	Administration of EP3 FP IP and TP agonists alone had no effect on
17574754	EP3	EP3	0.3	We investigated the effect of PGE 2 and the EP2 and EP3 receptors in vitro in a model of LPS
17574754	EP3	EP3	0.3	investigated the effect of PGE 2 and the EP2 and EP3 receptors in vitro in a model of LPS mediated inflammation
17574754	EP3	EP3	0.3	Selective activation of the EP2 and EP3 receptors also increased LPS-mediated CA1 neurotoxicity ( Fig
17574754	EP3	EP3	0.3	Selective activation of the EP2 and EP3 receptors also increased LPS-mediated CA1 neurotoxicity ( Fig 3 B
17574754	EP3	EP3	0.3	in vitro model and from our previous studies 3 the EP2 and EP3 receptors can also be excluded since they too
17574754	EP3	EP3	0.3	model and from our previous studies 3 the EP2 and EP3 receptors can also be excluded since they too rescue motor
17574754	EP3	EP3	0.3	Stimulation of the PGE 2 EP3 receptor with picomolar concentrations of agonist resulted in significant rescue
17574754	EP3	EP3	0.3	consistent with previous data obtained in spinal cord slices where EP3 activation rescued motor neurons subjected to chronic glutamate toxicity 3
17574754	EP3	EP3	0.3	rescued motor neurons subjected to chronic glutamate toxicity 3 here EP3 activation was associated with increased levels of the pro-survival phosphorylated
17574754	EP3	EP3	0.3	hippocampal slices activation of the EP2 15 and 16 and EP3 ( Fig 2 receptor rescues CA1 neurons stimulated with NMDA
17574754	EP3	EP3	0.3	cytokines and reactive oxygen species stimulation of the EP2 or EP3 receptor now increases LPS-mediated CA1 toxicity
17574754	EP3	EP3	0.3	nM the EP2 agonist butaprost (200 200 nM or the EP3 agonist sulprostone (10 10 nM consistently enhanced LPS-induced CA1 PI
17574754	EP3	EP3	0.3	The increased toxicity of LPS with addition of EP2/EP3 EP2 EP3 receptor agonists is relevant to recent in vivo studies demonstrating