HUGO ID Detailed Result 8975

PMID	Match String	Actual String	Score	Flanking text	Edited by	Edit
16104843	PI3K	PI3K	1.0	for endothelial cells from VEGFR-2 is reported to involve the PI3K (phosphoinositide phosphoinositide 3-kinase)/Akt 3-kinase Akt pathway 82 83 ( Figure
16104843	PI3K	PI3K	1.0	another pathway may be involved since the signal to activate PI3K by VEGFR-2 is usually not very strong
16104843	PI3K	PI3K	1.0	the activation of VEGFR-2 by VEGF-A results in the PI3K/Akt-dependent PI3K Akt-dependent activation of several integrins leading to enhanced cell adhesion
16104843	PI3K	PI3K-dependent	1.0	phosphorylation of VEGFR-3 has been shown to lead to a PI3K-dependent activation of Akt and PKC (protein protein kinase C -dependent
16104843	PI3K	PI3K	1.0	The activation of the PI3K/p70 PI3K p70 S6K (S6 S6 kinase pathway by VEGFR-2 is also
16104843	PI3K	PI3K	1.0	Furthermore the activation of eNOS is regulated by the PI3K/Akt PI3K Akt pathway 123 124
16104843	PI3K	PI3K	1.0	The small GTP-binding protein Rac which is also activated by PI3K has been implicated in the regulation of vascular permeability 125
16104843	PI3K	PI3K	1.0	neuropilin PAIP2 polyadenylated-binding protein-interacting protein 2 PDK1 phosphoinositide-dependent kinase 1 PI3K phosphoinositide 3-kinase PILSAP puromycin-intensive leucyl-specific aminopeptidase PKC protein kinase C
16647138	PI3	PI3-kinase	0.3	upregulates both endothelial nitric oxide synthase (eNOS) eNOS and PI3-kinase/Akt PI3-kinase Akt pathways	Junguk Hur
16647138	PI3	PI3-kinase	0.3	Not only eNOS inhibitors but also PI3-kinase/Akt PI3-kinase Akt signaling pathway inhibitors can prevent this upregulation by EPOX	Junguk Hur
16647138	PI3	PI3-kinase	0.3	This indicates that both eNOS and PI3-kinase/Akt PI3-kinase Akt pathways may mediate the angiogenic effects of EETs (	Junguk Hur
16647138	PI3	PI3-kinase	0.3	via a nitric-oxide-dependent mechanism as well as via activation of PI3-kinase and MARK pathways ( Wang et al. 2003 and Wang	Junguk Hur
16624536	phosphatidylinositol 3-kinase	phosphatidylinositol 3 kinase	1.0	through activation of the phosphatidylinositol 3 kinase/protein kinase b pathway fractalkine will inhibit fas ligand induced microglial apoptosis through down regulation of the pro apoptotic function of bad and up regulation of the anti apoptotic activity
16647138	pi3-kinase	pi3 kinase	1.0	in endothelial cell cultures the overexpression of epox upregulates both endothelial nitric oxide synthase enos and pi3 kinase/akt pathways.	Junguk Hur
16647138	pi3-kinase	pi3 kinase	1.0	not only enos inhibitors but also pi3 kinase/akt signaling pathway inhibitors can prevent this upregulation by epox.	Junguk Hur
16647138	pi3-kinase	pi3 kinase	1.0	this indicates that both enos and pi3 kinase/akt pathways may mediate the angiogenic effects of eets kawasaki et al. 2003 .	Junguk Hur
16647138	pi3-kinase	pi3 kinase	1.0	collectively these studies suggest that epox derived eets modulate angiogenesis via a nitric oxide dependent mechanism as well as via activation of pi3 kinase and mark pathways wang et al. 2003 and wang et al. 2005b .	Junguk Hur
16647138	phosphatidylinositol 3-kinase	phosphatidylinositol 3 kinase	1.0	paf stimulates phosphatidylinositol 3 kinase and mitogen activated protein map kinase and inhibits adenylate cyclase.
16753239	phosphatidylinositol 3-kinase	phosphatidylinositol 3 kinase	1.0	furthermore the studies demonstrated that 15d pgj 2 also altered astrocyte expression of these pro inflammatory molecules by inhibiting the phosphatidylinositol 3 kinase akt signaling pathway.
17350694	phosphatidylinositol 3-kinase	phosphatidylinositol 3 kinase	1.0	cascades; iii antioxidant activity mainly owing to the phenol group of various resorcinol type cannabinoids; iv suppression of the production of tumor necrosis factor _amp_#x3b1;; v activation of the phosphatidylinositol 3 kinase and protein kinase b pathway; vi induction of phosphorylation of extracellular regulated kinases; and vii induction of the expression of transcription factors and neurotrophins.