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| 15081582 | c-Jun | c-Jun | 0.3 | signals through blocking the activation of stress-activated protein kinase SAPK c-Jun N-terminal kinase (JNK) JNK ( Kawamura et al. 1999 | |  |
| 15210305 | c-Jun | c-jun | 2.4 | Immunohistochemical expression of a number of gene candidates including c-jun JNK/SAPK JNK SAPK (a a kinase that exerts a stress-related | | |
| 15210305 | c-Jun | c-jun | 2.4 | SAPK (a a kinase that exerts a stress-related activation of c-jun and NF-kappa B a transcription factor induced by oxidative injury | | |
| 17191135 | AP-1 | AP-1 | 1.6 | HO-1 gene such as Fos/Jun Fos Jun activator protein-1 (AP-1)], AP-1 nuclear factor-kB (NFkB) NFkB and the more recently identified Nrf2 | | |
| 17191135 | AP-1 | AP-1 | 1.6 | and WT-ZFP conferring constitutive expression inducible expression elements such as AP-1 AP-2 NF-kB Oct-1 PEA-3 Myb and the antioxidant-responsive element (ARE) | | |
| 17191135 | AP-1 | AP-1 | 1.6 | DNA binding activity of critical transcription factors such as the AP-1 family members NF-kB Jun Fos p53 CREB PEBP2/CBF, PEBP2 CBF | | |
| 15081582 | c jun | c jun | 1.0 | apoptotic death in nerve growth factor deprived pheochromocytoma pc12 cells by preventing the activation of apoptotic signals through blocking the activation of stress activated protein kinase sapk /c jun n terminal kinase jnk kawamura et al. 1999 . | | |
| 15210305 | c jun | c jun | 1.0 | c jun and zif 268 two immediate early genes factors widely considered as markers of neuronal response to injury and apoptotic cell death have been found to be markedly increased in dorsal and ventral horns | | |
| 15210305 | c jun | c jun | 1.0 | immunohistochemical expression of a number of gene candidates including c jun jnk/sapk a kinase that exerts a stress related activation of c jun and nf kappa b a transcription factor induced by oxidative injury with a prominent neuroprotective function have been assayed in different disease targeted tissues. | | |
| 15210305 | c jun | c jun | 1.0 | the jnk/sapk c jun pathway was found to be markedly up regulated [ 67 ] in als spinal cord astrocytes together with nf kappa b whereas motor neurones show a lower expression of these molecules. | | |