HUGO ID Detailed Result 3553

PMID	Match String	Actual String	Score	Flanking text	Edited by	Edit
16647138	FAAH	FAAH	2.2	the metabolism of AEA is fatty acid amide hydrolase (FAAH) FAAH
16647138	FAAH	FAAH	2.2	which is not a substrate for fatty acid amidohydrolase (FAAH), FAAH produced a similar increase in PGE 2 production as AEA
17350694	FAAH	FAAH	2.2	opening of intracellular Ca 2 stores inhibition of AMT and FAAH antioxidative effects
17350694	FAAH	FAAH	2.2	obtained through genetic ablation of fatty acid amide hydrolase (FAAH) FAAH 38 exert robust anti-inflammatory and neuroprotective effects in hSOD1G93A mice
17350694	FAAH	FAAH	2.2	Several studies have provided strong evidence that FAAH owing to its broad distribution might represent an attractive therapeutic
17350694	FAAH	FAAH	2.2	For example inhibition of FAAH by URB597 can augment endogenous brain levels of AEA and
17350694	FAAH	FAAH	2.2	Similarly AM374 another FAAH inhibitor has been shown to exert potent neuroprotective effects in
17350694	FAAH	FAAH	2.2	Furthermore another selective and powerful FAAH inhibitor that has been used for the treatment of pathological
17350694	FAAH	FAAH	2.2	on the endocannabinoid tone and thus favors the use of FAAH inhibitors to treat the inflammatory and neurodegenerative damage associated with
17350694	FAAH	FAAH	2.2	-acylphosphatidylethanolamine (NAPE)-specific NAPE -specific phospholipase D (NAPE-PLD) NAPE-PLD 59 and FAAH 60 and 61 with respect to AEA
17350694	FAAH	FAAH	2.2	AEA seems to be largely dependent on its hydrolysis by FAAH rather than on its synthesis by Ca 2 -dependent N
17350694	FAAH	FAAH	2.2	If not therapeutic agents per se inhibitors of NAPE-PLD FAAH AMT DAGL or MAGL could be used together with AEA
17350694	FAAH	FAAH	2.2	The mechanism of action of cannabidiol relies on AMT and FAAH inhibition and on antioxidative properties as a result this natural
17350694	FAAH	FAAH	2.2	up by cells AEA is a substrate for the hydrolase FAAH which breaks the amide bond and releases arachidonic acid and
17350694	FAAH	FAAH	2.2	Once accumulated in the cell 2-AG can be degraded by FAAH or more efficiently by a specific MAGL
17350694	FAAH	FAAH	2.2	the ECS are located in the plasma membrane apart from FAAH which is bound to intracellular membranes and MAGL which is
17350694	FAAH	FAAH	2.2	are able to modulate the AEA metabolic enzymes NAPE-PLD and FAAH might be exploited to curb neuroinflammation
16647138	fatty acid amide hydrolase	fatty acid amide hydrolase	1.0	the principal enzyme for the metabolism of aea is fatty acid amide hydrolase faah .
17350694	fatty acid amide hydrolase	fatty acid amide hydrolase	1.0	pharmacological agonists of cb receptors and increased levels of endocannabinoids obtained through genetic ablation of fatty acid amide hydrolase faah [38] exert robust anti inflammatory and neuroprotective effects in hsod1g93a mice delaying disease progression 39 and 40 .