HUGO ID Detailed Result 11917

PMID	Match String	Actual String	Score	Flanking text	Edited by	Edit
15572176	p75	p75	1.8	TrkA receptor it also can activate the non-selective neurotrophin receptor p75 NTR a member of the tumor necrosis factor receptor superfamily
15572176	NTR	NTR	0.5	receptor it also can activate the non-selective neurotrophin receptor p75 NTR a member of the tumor necrosis factor receptor superfamily	Junguk Hur
15572176	p75	p75	1.8	Signaling through p75 NTR in the absence of the corresponding Trk receptor has
15572176	NTR	NTR	0.5	Signaling through p75 NTR in the absence of the corresponding Trk receptor has been	Junguk Hur
15572176	p75	p75	1.8	Motor neurons express p75 NTR during the embryonic period of naturally occurring cell death
15572176	NTR	NTR	0.5	Motor neurons express p75 NTR during the embryonic period of naturally occurring cell death when	Junguk Hur
15572176	p75	p75	1.8	Although p75 NTR is not present in mature motor neurons the receptor
15572176	NTR	NTR	0.5	Although p75 NTR is not present in mature motor neurons the receptor can	Junguk Hur
15572176	p75	p75	1.8	Moreover p75 NTR is found in motor neurons of ALS patients 80
15572176	NTR	NTR	0.5	Moreover p75 NTR is found in motor neurons of ALS patients 80 and	Junguk Hur
15572176	p75	p75	1.8	receptor might modulate the death of neurons in damaged areas p75 NTR expression can be also observed on reactive astrocytes microglia
15572176	NTR	NTR	0.5	might modulate the death of neurons in damaged areas p75 NTR expression can be also observed on reactive astrocytes microglia and	Junguk Hur
15572176	p75	p75	1.8	Thus it is conceivable that NGF signaling between astrocytes and p75 NTR -expressing motor neurons may contribute to the induction of
15572176	NTR	NTR	0.5	it is conceivable that NGF signaling between astrocytes and p75 NTR -expressing motor neurons may contribute to the induction of neuronal	Junguk Hur
15572176	p75	p75	1.8	horn in symptomatic mice carrying the G93A mutation coincident with p75 NTR expression in motor neurons 93 suggesting that increased NGF
15572176	NTR	NTR	0.5	in symptomatic mice carrying the G93A mutation coincident with p75 NTR expression in motor neurons 93 suggesting that increased NGF production	Junguk Hur
15572176	p75	p75	1.8	Some isoforms of pro-NGF bind with high affinity to p75 NTR and thus induces a specific apoptotic signal even in
15572176	NTR	NTR	0.5	Some isoforms of pro-NGF bind with high affinity to p75 NTR and thus induces a specific apoptotic signal even in cells	Junguk Hur
15572176	p75	p75	1.8	induces a specific apoptotic signal even in cells expressing both p75 NTR and TrkA receptors 76
15572176	NTR	NTR	0.5	a specific apoptotic signal even in cells expressing both p75 NTR and TrkA receptors 76	Junguk Hur
15572176	p75	p75	1.8	recent reports further support a role for a NGF/p75 NGF p75 NTR apoptotic pathway in ALS
15572176	NTR	NTR	0.5	reports further support a role for a NGF/p75 NGF p75 NTR apoptotic pathway in ALS	Junguk Hur
15572176	p75	p75	1.8	with an antisense peptide nucleic acid construct that targets the p75 NTR gene and inhibits its expression 132
15572176	NTR	NTR	0.5	an antisense peptide nucleic acid construct that targets the p75 NTR gene and inhibits its expression 132	Junguk Hur
15572176	p75	p75	1.8	reported in double transgenic mice expressing SOD1 G93A but lacking p75 NTR 73
15572176	NTR	NTR	0.5	in double transgenic mice expressing SOD1 G93A but lacking p75 NTR 73	Junguk Hur
15572176	p75	p75	1.8	For example the re-expression of p75 NTR and neuronal NOS may help to determine which neurons
15572176	NTR	NTR	0.5	For example the re-expression of p75 NTR and neuronal NOS may help to determine which neurons survive	Junguk Hur
15572176	p75	p75	1.8	induce motor neuron apoptosis unless nitric oxide is produced or p75 NTR is expressed in those neurons
15572176	NTR	NTR	0.5	motor neuron apoptosis unless nitric oxide is produced or p75 NTR is expressed in those neurons	Junguk Hur
15572176	p75	p75	1.8	in motor neuron A which upregulates the expression of nNOS p75 NTR Fas cytokines and trophic factors
15572176	NTR	NTR	0.5	motor neuron A which upregulates the expression of nNOS p75 NTR Fas cytokines and trophic factors	Junguk Hur
16510725	TNFR2	TNF-R2	1.2	biological effects through activation of two distinct receptors TNF-R1 and TNF-R2 (Tartaglia Tartaglia and Goeddel 1992 alpha and its proapoptotic receptor
16624536	TNFR2	TNFR2	1.8	The interaction of TNF- or TNF-_amp_#x3b2 with TNFR2 leads to the activation of NF-_amp_#x3ba B and the transcription
16624536	TNFR2	TNFR2	1.8	Of interest knock out mice lacking TNFR2 show a failure to limit the immune response in experimental
16624536	TNFR2	TNFR2	1.8	autoimmune encephalitis and the use of p74 TNF receptor (TNFR2) TNFR2 antisense oligonucleotides will increase the extent of a hypoxic injury
16624536	p75	p75-dependent	1.8	been previously shown to induce motor neuron apoptosis via a p75-dependent mechanism 118 and 119
17015226	p75	p75	1.2	nitric oxide can induce apoptosis of motor neurons through the p75 NTR receptor
17015226	NTR	NTR	0.2	oxide can induce apoptosis of motor neurons through the p75 NTR receptor	Junguk Hur
17018025	p75	p75	1.2	with 2% horse serum and further incubated with mouse anti-rat p75 LNTR monoclonal antibody (p75, p75 1 800 Chemicon overnight at
17018025	p75	p75	1.2	further incubated with mouse anti-rat p75 LNTR monoclonal antibody (p75, p75 1 800 Chemicon overnight at 4_amp_deg C in blocking solution
17018025	p75	p75	1.2	As a specific marker for motoneurons p75 (the the low-affinity nerve growth factor receptor immunocytochemistry provides a
17018025	p75	p75	1.2	Motoneuron survival was assessed by direct counting of large p75 positive cells (cell cell bodies > 30 _amp_#x03BC m displaying
17418957	p75	p75	1.2	MNs by positive selection immuno-panning using antibody against the low-affinity p75 NGF receptor (192 192 antibody was a gift of the