HUGO ID Detailed Result 2615

PMID	Match String	Actual String	Score	Flanking text	Edited by	Edit
16647138	P450	P450	2.2	Cytochrome P450 epoxygenases (EPOX) EPOX
16647138	P450	P450	2.2	In addition to the COX and LOX pathways the cytochrome P450 (CYP450) CYP450 pathways also catalyze arachidonic acid conversion to biologically
16647138	P450	P450	2.2	Although the levels of various cytochrome P450 enzymes in brain are low it has been shown that
16647138	P450	P450	2.2	Studies on astroglial cytochrome P450 expression suggest a putative capacity of these enzymes to metabolize
16647138	P450	P450	2.2	most active steroidogenic cells in the brain expressing neurosteroidogenic cytochrome P450 and producing various neurosteroids
16647138	P450	P450	2.2	EETs and 20-HETE products of P450 arachidonic acid epoxygenase
16647138	P450	P450	2.2	Brain parenchymal tissue metabolizes arachidonic acid via the cytochrome P450 epoxygenase to epoxyeicosatrienoic acids (EETs), EETs which can dilate cerebral
16647138	P450	P450	2.2	Treatment of the donor vessels with a cytochrome P450 epoxygenase inhibitor (PPOH) PPOH eliminated dilator responses in both donor
16647138	P450	P450	2.2	Another P450 metabolite of arachidonic acid 20-HETE is a potent vasoconstrictor decreasing
16647138	P450	P450	2.2	Inhibition of P450 epoxygenase activity blocked the functional hyperemia in response to stimulation
16647138	P450	P450	2.2	The COX-2 inhibitor NS-398 was ineffective as were two cytochrome P450 antagonists (MS-PPOH MS-PPOH and miconazole suggesting that COX-2 and P450
16647138	P450	P450	2.2	P450 antagonists (MS-PPOH MS-PPOH and miconazole suggesting that COX-2 and P450 metabolites do not play a significant role in vasodilation as
16647138	P450	P450	2.2	evidence that the metabolites of arachidonic acid generated by cytochrome P450 epoxygenase play a significant role in the modulation of synaptic
16647138	P450	P450	2.2	Inhibitors of phospholipase A 2 and cytochrome P450 epoxygenase blocked this capacitative Ca 2 entry from the extracellular
16647138	P450	P450	2.2	2 as well as inhibitors of COX LOX and cytochrome P450 isozymes to reverse the depolarization
16647138	P450	P450	2.2	PLA 2 inhibitor 4-bromophenylacyl bromide (pBPB), pBPB or the cytochrome P450 inhibitor 17-octadecynoic acid (17-ODA), 17-ODA significantly restored the membrane potential
16647138	P450	P450	2.2	to the irreversible depolarization by in vitro ischemia with cytochrome P450 isozymes making a major contribution
16647138	P450	P450	2.2	EETs are the only cytochrome P450 metabolites of arachidonic acid produced by endothelial cells
16647138	P450	P450	2.2	(2002) 2002 reported that TIA induces the expression of P450 2C11 an arachidonic acid epoxygenase which is upregulated in the
16647138	P450	P450	2.2	metabotropic glutamate receptors are functionally linked to PLC and cytochrome P450 arachidonate epoxygenase activity ( Gebremedhin et al. 2003 indicating that
18513389	P450	P450	1.9	4 genotype excitotoxicity genes ciliary neurotrophic factor (CTNF), CTNF cytochrome P450 debrisoquine hydroxylase CYP2D6 apurinic apyrimidinic endonuclease (APEX), APEX mitochondrial manganese