HUGO ID Detailed Result 7661

PMID	Match String	Actual String	Score	Flanking text	Edited by	Edit
18219386	p67	p67	0.3	phox and recruitment of the activating cytosolic components p47 phox p67 phox and p40 phox are needed for function (Figure Figure	Junguk Hur
18219386	p67	p67	0.3	is phosphorylated thereby initiating translocation of the p47 phox /p67 p67 phox /p40 p40 phox complex to the membrane where phosphorylated	Junguk Hur
18219386	p67	p67	0.3	acts to coordinate the translocation of the p47 phox /p67 p67 phox /p40 p40 phox complex and is active only when	Junguk Hur
18219386	p67	p67	0.3	is believed to block the translocation of p47 phox /p67 p67 phox to Nox ( 23 and would therefore inhibit only	Junguk Hur
16877542	p67 phox	p67 phox	1.0	the levels of the p67 phox subunit that contains the nadph binding site of the nadph oxidase complex 10 were increased in membrane fractions of spinal cord extracts from symptomatic transgenic sod1 g93a mice fig 1 c indicating
18219386	p67 phox	p67 phox	1.0	nox2 is constitutively associated with the transmembrane nox stabilizing protein p22 phox and recruitment of the activating cytosolic components p47 phox p67 phox and p40 phox are needed for function figure 1 .
18219386	p67 phox	p67 phox	1.0	upon cell activation p47 phox is phosphorylated thereby initiating translocation of the p47 phox /p67 phox /p40 phox complex to the membrane where phosphorylated p47 phox binds to p22 phox .
18219386	p67 phox	p67 phox	1.0	rac acts to coordinate the translocation of the p47 phox /p67 phox /p40 phox complex and is active only when bound to gtp not when bound to gdp.
18219386	p67 phox	p67 phox	1.0	apocynin is believed to block the translocation of p47 phox /p67 phox to nox 23 and would therefore inhibit only the nox proteins requiring these cytosolic activators which include nox1 and nox2.
18219386	chronic granulomatous disease	chronic granulomatous disease	1.0	mutations altering the activity of nox2 or other subunits of nox are known to cause chronic granulomatous disease cgd 15 which is characterized by severe and chronic infections that are hard to treat due to the incapacity of immune cells to produce o 2 _amp_#x02022;_amp_#x02013; and therefore to fight pathogens.